This is an archived Horseadvice.com Discussion. The parent article and menus are available on the navigation menu below:
HorseAdvice.com » Diseases of Horses » Cardiovascular, Blood, and Immune System » Neonatal Isoerythrolysis »
  Discussion on Neonatal isoerythrolysis
Author Message
Member:
Mcgee

Posted on Sunday, Mar 13, 2005 - 10:13 pm:

Dr, O

I read your article on neonatal isoerytholysis and have a couple of questions. First, you said "when the foal of a SUBSEQUENT breeding inherits these antigens......" does that mean that the antigens originated in a PRIOR breeding and that stallion of this breeding might NOT be incompatible??

I had a healthy TB filly delivered four days ago. Yesterday I noted yellowing around the eyes and gums, although the filly is eating well and bucking and playing all of the time. The temp on the filly was 102.8 I called my vet and he took a blood sample this morning. He said the PCV was 24. This evening at 5pm he took another sample. I felt sure it would show improvement since the filly is still running, bucking and eating like normal. Unfortunately the PCV was down to 16 so he started antibiotics and cortisocoid steroids. We are taking another bloodf count at 9pm.. My vet has gotten blood donations from two geldings "just in case"

The filly seems less yellow in the eyes, yet her PCV is lower. Do you have any idea why?

My next question is this: would breeding her to a AQHA or warmblood(or paint etc) help this problem? She is a gorgeous, young, black TB mare about 17.2 hands and 1600 lbs so I am sure someone would love to breed her if she would be better off with a different breed of stallion.

She had one AQHA foal(no problem was related to me) before I purchased her. I bred her to one of my TB stallions. Do I dare try again, or do I need to blood test the stallions? Would an outcross breeding be better?

I feel lucky that the filly "acts" so normal, but why would her red blood cells be going down?

Thanks,

gail
Moderator:
DrO

Posted on Monday, Mar 14, 2005 - 8:01 am:

The mare develops the sensitivity to the antigens from the prior breeding but a effected foal of the present breeding has to have the antigens she has to become sensitized to. These antigens come from the father. These antigens are not specific to the individual but are found in the general equine population.

The lessening yellow may indicate a slower rate of breakdown, but they may still be breaking down which is why they continue to go down. You follow the PCV not the jaundice, its appearance is very subjective.

It does not have anything to do with the breed of the stallion gail, read the article more carefully as it goes into great depth as to how this happens and exactly how to prevent it from happening again.

Be sure the foal is getting enforced rest during this.
DrO
Home Page | Top of Page | Join Us!
Horseadvice.com is a BBB Accredited Business. Click for the BBB Business Review of this Horse Training in Stokesdale NC